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He received his medical degree in 1982 and his Ph.
Whilst impressive clinical benefits have been observed for NSCLC with a known driver mutation, acquired resistance is frequently seen and presents us with the next challenge in the goal to deliver unique personalised medicine.
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The most robust data is seen in the six trials that evaluated either erlotinib or gefitinib (epidermal growth factor receptor (EGFR) tyrosine kinase inhibitors (TKIs)) as switch maintenance.
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In patients with resistance to first-generation EGFR TKIs generated by MET amplification, it is unlikely that an irreversible EGFR inhibitor alone would be effective, but the combination of an irreversible EGFR inhibitor and an mTOR inhibitor may be an effective strategy for overcoming resistance ().
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This correlates with our observation that these clones exhibit the highest levels of GTP-bound KRAS ().
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Conclusions: Preoperative PET-CT may detect cases of otherwise subclinical spread to N2 glands, distant metastasis, or extensive, nonresectable local disease, thereby avoiding major surgical procedures in such non-curable disease.
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The aim of the present work was to examine the effects of a combined treatment of metformin with gefitinib, a selective EGFR-TKI on NSCLC cell lines.
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In May 2011, the USFDA approved Sunitininb for treating patients with 'progressive neuroendocrine cancerous tumors located in the pancreas that cannot be removed by surgery or that have spread to other parts of the body (metastatic)'.
The simulation process was conducted in a sequential manner, initially using the PK model to generate the AUC t values for each selected dose regimen, which subsequently defined the PD variables (IC 50 and K tr) via the functional equations (see Supplementary Fig.
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Two hits are better than one: targeting both phosphatidylinositol 3-kinase and mammalian target of rapamycin as a therapeutic strategy for acute leukemia treatment.
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Reversing Warburg effect might be a profound strategy for cancer treatment and is becoming a hot research area.
TLG-S criteria are superior to both EORTC and PERCIST for predicting outcomes in patients with metastatic lung adenocarcinoma treated with erlotinib.
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Debido a que este es muy probablemente el resultado de una reducción en el error de estadificación, es decir, una disminución de la probabilidad de pasar por alto los ganglios linfáticos positivos con un número cada vez mayor de muestras de ganglios linfáticos, se indica que una evaluación del estado ganglionar debe incluir entre 11 y 16 ganglios linfáticos.
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These results suggested that crosstalk between ectopic human EGFR and endogenous signaling proteins may occur in the transgenic mice.
Therapy was continued until disease progression, concomitant illness preventing further administration, unacceptable toxicity, or patient withdrawal.
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The relationship between EGFR mutation and clinical outcome was examined by Fisher's exact test.
However, consistent with the notion that erlotinib is more effective against EGFR-mutant tumors, a recent study reported that in patients with NSCLC harboring wild-type EGFR, docetaxel induced a higher RR (13.
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Doxorubicin activates FOXO3a to induce the expression of multidrug resistance gene ABCB1 (MDR1) in K562 leukemic cells.
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EGF receptor gene mutations are common in lung cancers from “never smokers” and are associated with sensitivity of tumors to gefitinib and erlotinib.
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Similar to ALK FISH, IHC requires one unstained slide cut from an FFPE block as long as there are at least a few clusters of viable tumor cells.
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In patients older than 74 years with stage IA NSCLC, Okami et al found no significant difference in 5-year survival after sublobar resection versus standard lobectomy, although locoregional recurrence rates were higher after sublobar resection.
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Comparison of the EGFR resistance mutation profiles generated by EGFR targeted tyrosine kinase inhibitors and the impact of drug combinations.
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Preoperative gefitinib versus gefitinib and anastrozole in postmenopausal patients with oestrogen-receptor positive and epidermal-growth-factor-receptor-positive primary breast cancer: a double-blind placebo-controlled phase II randomised trial.
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